Preliminary studies reveal conolidine may possibly inhibit distinct ion channels, minimizing neuronal excitability and limiting soreness signals. This mechanism is particularly appropriate in neuropathic pain, exactly where abnormal signaling causes persistent pain. In addition, conolidine seems to influence G protein-coupled receptor (GPCR) pathways integral to agony perception. Comp... https://davidn617wfi0.wikikali.com/user